A lyophilized solid for stability, Semax 5mg is a high-purity research peptide. It is investigated in models of neuroprotection, cognitive enhancement, and immune support after being verified at >99.1% purity (HPLC-tested) with complete COA provided. solely for use in research.
Semax is a synthetic derivative of adrenocorticotropic hormone (ACTH) widely studied in preclinical and laboratory models for its potential cognitive-enhancing, neuroprotective, and mood-supportive effects. Supplied in lyophilised (freeze-dried) form to preserve stability, this compound is verified at >99.1% purity (HPLC-tested) and provided with full Certificates of Analysis (COAs).
Semax is a peptide analogue derived from ACTH fragments, developed to extend neurotropic activity without hormonal side effects. Research has investigated its influence on brain-derived neurotrophic factor (BDNF), synaptic plasticity, and vascular health mechanisms. Unlike consumer supplements, Semax supplied by Bluewell Peptides is strictly intended for laboratory research use only and comes with full COA verification for transparency.
Product Name: Semax 5mg
Catalogue Number: BWP-SMX-5
CAS Number: 80714-61-0
Molecular Formula: C₃₇H₅₁N₉O₁₀S
Molecular Weight: 813.92 g/mol
Form: Lyophilised Solid
Purity: >99.1% (HPLC Verified)
Storage: Store at −20°C, protected from light
Unit Size: 5mg
Semax is supplied as a lyophilised solid to ensure long-term stability. For laboratory use, it may be reconstituted with bacteriostatic water or another suitable solvent according to established protocols. For detailed guidance, please refer to our peptide reconstitution page.
Semax has been investigated in preclinical and laboratory studies for potential roles in:
Cognitive Enhancement and Neuroprotection – Studied for its ability to increase BDNF levels, supporting learning, memory, and synaptic function.
Immune and Cardiovascular Support – Researched for its potential to regulate immune responses and enhance vascular health.
Pain and Inflammation Modulation – Preliminary studies suggest roles in reducing pain perception and regulating inflammatory pathways.
References available on request or through our research archive.
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